Hyperammonaemia is a TIME CRITICAL medical emergency with the risk of death and serious neurological damage.
As we know “TIME is BRAIN” so early recognition of stroke is vital so BE FAST! Read more
Hyperammonaemia is a TIME CRITICAL medical emergency with the risk of death and serious neurological damage.
Tips:


Information from APLS Aide-Memoire
The Yorkshire & Humber Paediatric Critical Care ODN, has released some useful guidance about the Post-ROSC phase of care 

PUT OUT A NEONATAL CRASH CALL
CRH – will result in at least a Neonatal SpR, SHO +/- a neonatal nurse.
HRI – may not generate a response. Consider a crash bleep to anaesthetics
CALL NEONATAL CONSULTANT (WILL ONLY GIVE ADVICE AS AT CRH)
CALL THE ED CONSULTANT
***Remember Grab Box***
Unless within the first few hours of life using the APLS algorithm is equally if not more appropriate

Paediatric Hypoglycaemia (BM <2.6) is a relatively common presentation in the Emergency Department. However, if we don’t do the BM it’s easy to miss.
Hypoglycaemia in paediatric diabetic patients is managed separately – see here
Hypoglycaemia in neonates (<72 hours of life) also has specific management – see here (Flowcharts A + B)
Hypoglycaemia is generally caused by disruption in one of the following:
Diabetic Ketoacidosis – remember in paediatrics this may be the 1st presentation of diabetes.
DKA Management Flow Chart – HERE
Diabetic children sometimes attend ED with hyperglycaemia, but not in DKA (what should we do?)
Paeds have produced some advice to follow:
To clarify the immobilisation strategy for patients requiring Poly-Trauma CT Scans (anything more than an isolated CT Head)
As there are now no longer a paediatric clinical team at HRI, the paediatric surgical pathway is up-dating, this is the current provisional pathway, to ensure care falls inline with GIRFT report.